<?xml version="1.0" encoding="UTF-8"?>
<compound>
  <version>1.0</version>
  <creation_date>2016-05-25 22:19:28 UTC</creation_date>
  <update_date>2026-04-05 15:57:33 UTC</update_date>
  <accession/>
  <common_name>Disulfides</common_name>
  <class>SmallMolecule</class>
  <description>Disulfides, with the chemical formula H2S2 and an average molecular weight of 66.15 g/mol, is a chemical compound found in various industrial and consumer applications. Disulfides belongs to the other non-metal sulfides, a subclass of other non-metal organides within the inorganic compounds.

The compound is associated with 22 recorded sources from which it is found in or released. Within the category of electrical and electronic equipment, these sources include antennas, auxiliary devices, communication cables or conductors, contacts, and adjustable resistors. In the sector of food, food processing and cookware, the compound is linked to the treatment of hops, the processing of meats, and the preparation of wine or sparkling wine and vinegar. Additionally, disulfides is found in household cleaning and consumer products, specifically in tobacco product cases and pipe accessories. Finally, it is recorded in building and construction materials, specifically in stairs and ramps.</description>
  <categories/>
  <types>
    <type>Human Neurotoxin</type>
  </types>
  <synonyms/>
  <chemical_formula>H2S2</chemical_formula>
  <average_molecular_weight>66.146</average_molecular_weight>
  <monisotopic_moleculate_weight>65.959791444</monisotopic_moleculate_weight>
  <iupac_name>dithioperoxol</iupac_name>
  <traditional_iupac>disulfane</traditional_iupac>
  <cas_registry_number>13465-07-1</cas_registry_number>
  <smiles>SS</smiles>
  <inchi>InChI=1S/H2S2/c1-2/h1-2H</inchi>
  <inchikey>BWGNESOTFCXPMA-UHFFFAOYSA-N</inchikey>
  <taxonomy>
    <kingdom>Inorganic compounds</kingdom>
    <super_class>Homogeneous non-metal compounds</super_class>
    <class>Other non-metal organides</class>
    <sub_class>Other non-metal sulfides</sub_class>
    <direct_parent>Other non-metal sulfides</direct_parent>
    <alternate_parents>
      <alternative_parent>Inorganic sulfides</alternative_parent>
    </alternate_parents>
    <substituents>
      <substituent>Inorganic sulfide</substituent>
      <substituent>Other non-metal sulfide</substituent>
    </substituents>
    <molecular_framework/>
    <descriptors>
      <descriptor>inorganic disulfide</descriptor>
      <descriptor>sulfur hydride</descriptor>
    </descriptors>
  </taxonomy>
  <status>Detected and Not Quantified</status>
  <origin/>
  <cellular_locations/>
  <biofluid_locations/>
  <tissue_locations/>
  <pathways>
    <pathway>
      <name>Purine Metabolism</name>
      <smpdb_id>SMP0000050</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Adenosine Deaminase Deficiency</name>
      <smpdb_id>SMP0000144</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Adenylosuccinate Lyase Deficiency</name>
      <smpdb_id>SMP0000167</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Gout or Kelley-Seegmiller Syndrome</name>
      <smpdb_id>SMP0000365</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Lesch-Nyhan Syndrome (LNS)</name>
      <smpdb_id>SMP0000364</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Molybdenum Cofactor Deficiency</name>
      <smpdb_id>SMP0000203</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Xanthine Dehydrogenase Deficiency (Xanthinuria)</name>
      <smpdb_id>SMP0000220</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Purine Nucleoside Phosphorylase Deficiency</name>
      <smpdb_id>SMP0000210</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>AICA-Ribosiduria</name>
      <smpdb_id>SMP0000168</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Azathioprine Action Pathway</name>
      <smpdb_id>SMP0000427</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Mercaptopurine Action Pathway</name>
      <smpdb_id>SMP0000428</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Thioguanine Action Pathway (old)</name>
      <smpdb_id>SMP0000430</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Xanthinuria Type I</name>
      <smpdb_id>SMP0000512</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Xanthinuria Type II</name>
      <smpdb_id>SMP0000513</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Adenine Phosphoribosyltransferase Deficiency (APRT)</name>
      <smpdb_id>SMP0000535</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Mitochondrial DNA Depletion Syndrome-3</name>
      <smpdb_id>SMP0000536</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Myoadenylate Deaminase Deficiency</name>
      <smpdb_id>SMP0000537</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Adenosine Deaminase Deficiency</name>
      <smpdb_id>SMP0125721</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Adenylosuccinate Lyase Deficiency</name>
      <smpdb_id>SMP0125722</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Gout or Kelley-Seegmiller Syndrome</name>
      <smpdb_id>SMP0125723</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Molybdenum Cofactor Deficiency</name>
      <smpdb_id>SMP0125725</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Xanthinuria Type I</name>
      <smpdb_id>SMP0125727</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Purine Nucleoside Phosphorylase Deficiency</name>
      <smpdb_id>SMP0125728</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Xanthinuria Type II</name>
      <smpdb_id>SMP0125731</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Adenine Phosphoribosyltransferase Deficiency (APRT)</name>
      <smpdb_id>SMP0125732</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Mitochondrial DNA Depletion Syndrome-3</name>
      <smpdb_id>SMP0125734</smpdb_id>
      <kegg_map_id/>
    </pathway>
    <pathway>
      <name>Myoadenylate Deaminase Deficiency</name>
      <smpdb_id>SMP0125735</smpdb_id>
      <kegg_map_id/>
    </pathway>
  </pathways>
  <state/>
  <appearance/>
  <experimental_properties/>
  <predicted_properties>
    <property>
      <kind>logp</kind>
      <value>1.04</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>pka_strongest_acidic</kind>
      <value>9.23</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>iupac</kind>
      <value>dithioperoxol</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>average_mass</kind>
      <value>66.146</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>mono_mass</kind>
      <value>65.959791444</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>smiles</kind>
      <value>SS</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>formula</kind>
      <value>H2S2</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>inchi</kind>
      <value>InChI=1S/H2S2/c1-2/h1-2H</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>inchikey</kind>
      <value>BWGNESOTFCXPMA-UHFFFAOYSA-N</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>polar_surface_area</kind>
      <value>0</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>refractivity</kind>
      <value>13.11</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>polarizability</kind>
      <value>6.02</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>rotatable_bond_count</kind>
      <value>0</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>acceptor_count</kind>
      <value>0</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>donor_count</kind>
      <value>2</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>physiological_charge</kind>
      <value>0</value>
      <source>ChemAxon</source>
    </property>
    <property>
      <kind>formal_charge</kind>
      <value>0</value>
      <source>ChemAxon</source>
    </property>
  </predicted_properties>
  <route_of_exposure/>
  <mechanism_of_toxicity/>
  <metabolism/>
  <toxicity/>
  <lethaldose/>
  <carcinogenicity/>
  <use_source/>
  <min_risk_level/>
  <health_effects>#&lt;HealthEffect::ActiveRecord_Associations_CollectionProxy:0x00007f7222bcc8c0&gt;</health_effects>
  <symptoms/>
  <treatment/>
  <drugbank_id/>
  <hmdb_id/>
  <pubchem_compound_id>108196</pubchem_compound_id>
  <chembl_id/>
  <chemspider_id>97274</chemspider_id>
  <kegg_id/>
  <uniprot_id/>
  <omim_id/>
  <chebi_id/>
  <biocyc_id/>
  <ctd_id/>
  <stitch_id/>
  <actor_id/>
  <pdb_id/>
  <wikipidia/>
  <general_references/>
  <targets>
    <target>
      <target_id/>
      <mechanism_of_action/>
      <constants/>
      <references/>
    </target>
  </targets>
</compound>
